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GHRH analog● Clinical trials

Tesamorelin

Tesamorelin is a synthetic GHRH (growth-hormone-releasing hormone) analog approved by the FDA as Egrifta/Egrifta SV/Egrifta WR for reduction of excess visceral abdominal fat in HIV-infected adults with lipodystrophy. It works by stimulating the pituitary to release growth hormone, which lowers visceral adipose tissue without a general weight-loss effect.

Molecular weight
~5135.8 g/mol
g/mol
Half-life
~8-11 min after SubQ dosing
estimated
Form
Lyophilized powder for reconstitution, SubQ (Egrifta SV/WR, Rx only)
FDA status
FDA-approved (Egrifta / Egrifta SV / Egrifta WR) for excess abdominal fat in HIV-associated lipodystrophy; not indicated for general weight loss

Tesamorelin reconstitution calculator

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Typical dosing

GoalRangeRouteFrequency
HIV-associated lipodystrophy (label, Egrifta SV)1.4 mgSubQ (abdomen, rotate sites)once daily
HIV-associated lipodystrophy (label, Egrifta WR)1.28 mgSubQ (abdomen, rotate sites)once daily

Ranges compiled from research literature — not a prescription.

How it works

Tesamorelin is a 44-amino-acid analog of native GHRH, modified with an N-terminal trans-3-hexenoic acid group that resists enzymatic (DPP-4) degradation and extends its activity relative to endogenous GHRH. It binds GHRH receptors on pituitary somatotroph cells, a Gs-coupled pathway that raises cAMP and triggers calcium-dependent, pulsatile release of growth hormone — mirroring the body's natural GH secretion pattern rather than delivering GH directly.

The downstream GH pulses raise circulating IGF-1 and, in HIV-infected patients with abdominal fat accumulation, produce a measurable reduction in visceral adipose tissue (VAT). This is the basis of the only approved indication: a pooled analysis of two 26-week phase 3 trials (n=806) reported meaningful VAT reduction with tesamorelin 2 mg/day versus placebo, and a separate randomized trial (Stanley et al., JAMA 2014) found tesamorelin reduced VAT by roughly 9.9% versus a 6.6% increase with placebo, along with modest reductions in liver fat, without worsening glucose or lipid parameters in that population.

It is important to be precise about scope: the FDA approval and this trial evidence are specific to HIV-associated lipodystrophy — a distinct metabolic condition tied to antiretroviral therapy and HIV infection itself. Use of tesamorelin for general anti-aging, bodybuilding, or fat loss in people without HIV lipodystrophy is off-label and has not been evaluated in the same rigorous, FDA-reviewed trials; efficacy and safety in that population are not established by the approved label.

Because tesamorelin acts upstream (on the pituitary) rather than supplying GH directly, its effects depend on an intact hypothalamic-pituitary axis — which is also why pituitary axis disruption is a contraindication, and why its GH/IGF-1 stimulating effect carries the same theoretical long-term considerations (e.g., malignancy risk, IGF-1-related effects) associated with other GH-axis therapies.

Side effects & safety

  • Fluid retention is common and can present as edema, arthralgia, myalgia, or carpal tunnel syndrome, especially early in treatment.
  • Because tesamorelin raises IGF-1, the FDA label calls for monitoring IGF-1 levels during therapy and considering discontinuation with persistent elevation, given unknown long-term effects of sustained elevation.
  • Glucose intolerance can occur; the label recommends evaluating glucose before and during treatment, with added caution in patients with diabetes or prediabetes.
  • Contraindicated in active malignancy — pre-existing malignancy must be inactive and treatment complete before starting.
  • Contraindicated in pregnancy and in patients with disruption of the hypothalamic-pituitary axis (pituitary surgery, radiation, tumor) or hypersensitivity to tesamorelin/excipients.
  • Injection-site reactions and paresthesias are among the most common adverse effects; this profile and all dosing above reflect the FDA-approved use only — it does not extend to unapproved (off-label) uses.

Research

Primary sources — PubMed.

  1. FDA. EGRIFTA SV (tesamorelin for injection) Prescribing Information. https://www.accessdata.fda.gov/drugsatfda_docs/label/2019/022505s012s013lbl.pdf
  2. DailyMed. EGRIFTA SV label. https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=3d783378-b02d-4f19-99dd-0fc91a042224
  3. Falutz J, et al. Effects of tesamorelin on visceral fat: pooled analysis of two phase 3 trials. J Clin Endocrinol Metab. 2010. https://academic.oup.com/jcem/article-abstract/95/9/4291/2835394
  4. Stanley TL, et al. Effect of Tesamorelin on Visceral Fat and Liver Fat in HIV-Infected Patients. JAMA. 2014. https://pmc.ncbi.nlm.nih.gov/articles/PMC4363137/
  5. Tesamorelin. LiverTox, NCBI Bookshelf. https://www.ncbi.nlm.nih.gov/books/NBK548730/
  6. ClinicalTrials.gov. TH9507 in Patients With HIV-Associated Lipodystrophy. NCT00123253. https://clinicaltrials.gov/study/NCT00123253
⚠︎Tesamorelin may be a research chemical not approved for human use in your country. This page is educational information only — not medical advice, and not an endorsement to use it.
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Tesamorelin FAQ

Reduction of excess visceral abdominal fat in HIV-infected adults with lipodystrophy, and nothing else. Marketed as Egrifta, Egrifta SV, and Egrifta WR, it is prescription-only. HIV-associated lipodystrophy is a distinct metabolic condition tied to antiretroviral therapy and HIV infection itself, so the approval does not extend to general obesity, bodybuilding, or anti-aging use.
No, it redistributes fat rather than reducing overall weight. It specifically lowers visceral adipose tissue: a randomized trial by Stanley et al. found a roughly 9.9% VAT reduction versus a 6.6% increase on placebo, with modest liver fat reductions and no worsening of glucose or lipid parameters in that population. It is not indicated or validated as a weight-loss drug.
Its half-life is only about 8-11 minutes after subcutaneous dosing. Tesamorelin acts upstream on pituitary GHRH receptors to trigger the body's own pulsatile growth hormone release rather than supplying GH directly, so each dose produces a discrete pulse. That mechanism also means it depends on an intact hypothalamic-pituitary axis, which is why axis disruption is a contraindication.
Yes. Because it raises IGF-1, the FDA label calls for monitoring IGF-1 levels during therapy and considering discontinuation with persistent elevation, given unknown long-term effects. Glucose should be evaluated before and during treatment, with added caution in diabetes or prediabetes. It is contraindicated in active malignancy, in pregnancy, and with hypothalamic-pituitary axis disruption.

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